Wound care essentials summative assignment

Wound c atomic number 18 essentials summative assignmentModule reckon Wound C ar Essentials plane section 1. Search schemeDescribe the strategy you utilise to retrieve the right re comes to help you write your assignment. You must take the key words you occasiond, the sources of your literature, the years searched and the character reference of literature you were looking for.Harvard (2007) verbalise that a well-structured literature search is an legal way to get authentic evidence on the topic being searched. The intended sources that depart be use atomic number 18 health addressfulness databases, governing bodyatic review libraries and specialist organisations.In searching, keywords were created using Boolean logics AND, OR, NOT and phrases with quotations. The fol natural depressioning ar the list of keywords innovation ulceration* CINAHL- retrieved 1842 records-COCHRANE lib retrieved 3 records PubMed retrieved- 1770 recordsdiabetic al-Qaida ulcer* CINAHL- retrieved 844 recordsCOCHRANE 2 records Cochrane reviewsPubMed 46 recordsdiabetes mellitus* CINAHL 54940 recordsPubMed 32863 recordsCOCHRANE 0 recorddiabetic neuropathy* CINAHL 1003 recordsPubMed 802 recordsCOCHRANE 0IJDDC 76 records sagaciousness woodpecker* AND diabetic bag ulcer* CINAHL 24 bequeathsPubMed 2 resultsCOCHRANE 0 supranational Journal of Diabetes in Developing Countries 0 manage* AND diabetic al-Qaida ulcer* CINAHL 216 resultsPubMed 24 resultsCOCHRANE 0IJJDC 0manage* OR handling* AND diabetic clean ulcer* CINAHL 224573 resultsPubMed 24 resultsCOCHRANE 1131 resultsIJJDC 0prevalence AND diabetic metrical unit ulcer*- CINAHL 64 resultsPubMed 9 recordsCOCHRANE 0 recordIJJDC 11 recordstreatment* OR intervention* AND diabetic posterior ulcer* CINAHL 316806PubMed 33 recordsCOCHRANE 39911 recordsIJDDC 5 recordscost* AND diabetic foot ulcer* CINAHL 97 recordsPubMed 4 recordsCOCHRANE 0evidence base* AND diabetic foot ulcer* CINAH L 73 recordsPubMed 1 recordCOCHRANE 0 dole out guideline* AND diabetic foot ulcer* CINAHL- 3 recordsCOCHRANE 0PubMed 0Eligible studies were place by searching the Cochrane Library (November 8, 2010), Cochrane Wound assort (2000 to November foremost week 2010), PubMed (2000 to November 1st week 2010), EBSCO CINAHL plus (2000 to November 2nd week 2010). Furthermore, reliable websites and links were apply in addition such as www.intute.ac.uk, www.boperis.ac.uk, www.dh.gov.uk, www.library.nhs.uk, Wound Cargon Alliance, World tolerant Wounds Electronic Journal, the Tissue Vi exponent Society, and the external Journal of Diabetes in Developing Countries or www.ijddc.com.Peer reviewed and published journals were employ which are reliable and reviewed by round separate(prenominal) authors. Thither was no restriction on language of publications. All publications were no more than 10 years old.Section 2. Wound aetiology use up a uncouth break type (e.g. diabetic foot ulcer ation, storm ulcer, pin ulcer, fungating offending, dehisced surgical provoke), which you moderate get byd for in your role as a qualified nurse. It may help to reflect on a ache-suffering you bemuse cared for with this type of wound. use contemporary literature to support your work, disputeWhat your chosen wound type isHow this type of wound develops (including contributory factors)How this type of wound is recognised (common characteristics)Who it affectsPrevalence in UK and home country rocky word count 800_______________________________________________________________________________Your fare hereOne of the common types of wounds is diabetic foot ulcer. International Working Group on the diabetic introduction (2010) defined diabetic foot ulcer in their research system as a liberal-thickness penetration of the dermis of the foot in a person with diabetes. According to Jeffocoate and Harding (2003) diabetic foot ulceration is a common complication of diabetes and it is disabling and frequently leads to leg amputation. It usu tout ensemble(a)y occurs as a result of neuropathic, vascular changes of a diabetic foot, foot deformities, plantar hardness and smoking (NICE, 2010).Even though the cause of diabetic peripheral neuropathy is uncertain, it is cognise that the nerve amour of a diabetic long-suffering degenerates in result to metabolic changes, pressure and ischaemia (Alexander, Fawcett Runciman, 2000). On the other hand, the carriage of mel minuscule sugar level in the blood such as sorbitol brush off cause osmotic puffiness and subsequent damage to the nerve cell, ontogenesis the risk of vascular disease and git too give bound to neuropathy and increase the risk of contagion (Falanga, 2005 Watkins, 2000). Pendsey (2010) stated that the neuropathy in diabetic patients is manifested in the motor, autonomic and sensory components of the nervous system.In motor neuropathy, the innervations of the intrinsic foot muscles are dama ged that leads to an imbalance between flexion and extension of the diabetic foot. It besides affects the muscles inevitable for normal foot movement altering the dissemination of forces during walking. This creates anatomic foot deformities that make abnormal bony prominences and pressure points and make sputter reactive callus at the sites of abnormal load. It then step by step causes skin breakdown and ulceration (Pendsey, 2003).In sensory neuropathy, it affects the peripheral sensation, after loss of sensation that makes the patient unable to feel trauma to their set about extremities. Patient leave alone too feel heaviness, insensitivity to heat, cold and pressure. It is estimated that 45-60% of all diabetic ulcerations are due sensory neuropathy (Frykberg, Zgonis, Armstrong et al., 2006). According to Alexander et al (2000) when mechanical forces continue to be applied on the affected area, it leads to inflammation, abscess organic law and, eventually, ulceration. Thi s is also the reason many wounds go unnoticed and get worse progressively since the affected area is continuously subjected to repetitive pressure and soak forces from ambulation and weight bearing without noticing it.In autonomic neuropathy the peripheral nerve function is affected, which controls the distribution of blood through arteriolar vessels. One of the signs and symptoms is decreased fret in the lower extremities which make the skin becomes dry and change magnitudely accustomed to fissures (Alexander et al. 2000).Moreover, poor blood supply to the foot or ischaemia is another significant risk factor for diabetic foot ulceration, which according to Frykberg et al. (2006) it much occurs in combination with loss of sensation and the researchers also said that an estimated 45% of diabetic ulcers are due to ischaemia and neuropathy.diabetic foot ulcer is commonly found in areas where the abnormal pressure distribution arises from humiliated foot architecture. It is usua lly located on hallux, graduation exercise metatarsal and twenty percent metatarsal heads, and under the heel (Grey, Enoch Harding, 2006). The precipitating causes of foot ulceration and transmittance are friction in ill fitting or rising shoes, untreated or self treated callus, foot injuries, burns, corn plaster, fill out infections and heel friction in patients confined to bed (Watkins, 2003).According to International Working Group on the Diabetic Foot (IWGDF) diabetes is global epidemic with devastating human, social and economic consequences. The disease claims many lives and places a dread(a) burden on healthcare systems and economies everywhere, with the heaviest burden falling on low and middle income countries such as Philippines. It is estimated that 250 million people worldwide drive home diabetes, equating roughly 6% of the heavy(p) population with the age group 20-79 years. The number is expected to reach about 380 million by 2025, representing 7.1% of the adu lt population. This is due to the current lifestyles which is lack of exercise and not having a tight-laced diet.A survey conducted by Morgan, Currie, Smithers, Butler and Peters (2000) revealed that foot problems occur in nearly 20% of diabetic patients. Two-thirds of lower extremity amputations are performed in diabetic patients alone, and a majority of these are preceded by a foot ulcer. Nwabudike and Ionescu-Tirgoviste (2008) set out a study to identify the clinical parameters associated with foot ulceration in diabetic patients. The study suggested that older age diabetic patient poses the risk of developing foot ulcer because of decreased ability to self care, including personal daily foot examinations. This is also a result of poor vision and imp distributeed mobility which also predispose patients to trauma. The study also shows that most patients with ulcer were type 2 diabetic patients and this correlates with the age of the patient group. The average duration of diabetes the patients have was of 11.5 years. It is twice more common in males may be due to the active nature of the activities that men are engaged compared to women, increasing the likelihood they may suffer from trauma. In Nwabudike (2008) research, it also showed that lack of awareness of the disease process and personal care increasing the likelihood that the ulcers may degenerate and leads to gangrene and amputation.Section 3. Wound measure outmentIdentify features of your chosen wound aetiology (wound type) that are commonly identified during the assessment process. Now select one of these features and critically discuss variant ways of assessing this problem. You must link your interchange to the contemporary wound care literature.Here is a list of wound features commonly identified during assessmentOdourExudateInfectionWound bed thread e.g. slough, necrotic windYour discussion must make clear which aspect of wound assessment you have chosen and involve an exploration of the different options visible(prenominal) for measuring, describing and entrying it.Approximate word count 500Your serve well here consummate wound assessment is essential for the appropriate and realistic planning of goals and interventions for patients with wounds (Collier, 2003).Diabetic foot wound has two classifications, the neuropathic foot ulcer and neuroischemic foot ulcer. Differentiating between these entities is essential because their complications are different and they require different therapeutic strategies (Pendsey, 2007). Neuropathic wound has no sensation and foot is warm to touch with intact pulses. The ulcerations are usually located on tips of toes and plantar surfaces under metatarsal heads. It also shows oedema, local necrosis and sepsis. On the other hand, ischaemic ulcer is dreadful upon rest and usually diminished sensation over period of time. Moreover, the foot is not warm to touch and has no pulse. The ulcerations are often located on margins of foot in particular on the medial surface of the first metatarsophalangeal joint and over the lateral aspect of the fifth metatarsophalangeal joint. They also develop on the tips of the toes and heels. Signs of sepsis, necrosis or gangrene are also noted.An schematic clinical tool TIME which is adapted from Watret (2005) is being apply for assessing the wound bed. The acronym stands for waver, infection, moisture balance and advancing or undermining epithelium.Necrotic create from raw stuff, slough and eschar are non-viable tissues that can be found on diabetic ulcer that inevitably to be removed through debridement while the presence of epithelial tissue and granulation tissue in the wound suggests healing. The unhealthy granulation tissue often dark in colour and frequently bleeds on mite are signs of infection. Moisture in the wound bed needs to be assessed too. Moist is known to stimulate healing by promoting granulation and load-bearing(a) debridement, neverthe slight, moistur e balance should be maintained to prevent the wound from get too dry and too moist which could stand up wound healing. In addition the wound edges and environs ordain be assessed for migrating epithelial cells which is a good sign of healing or maceration which suggest poor care.Infection is a major factor that affects the time healing of all wounds. Jeffcoate and Harding (2003) said that infection can cause substantial deterioration and delay wound healing. Infection is an invasion and growth of pathogenic microorganisms in the body. Diabetic foot infection is divided in three categories superficial and local, flocculent tissue and spreading or cellulitis, and osteomyelitis (Jeffcoate Harding, 2003). The classic signs of infection are heat, redness, swelling and pain. Other signs such as increase exudates, delayed healing, odour, and abnormal granulating tissue are also suggesting infection (Grey et al, 2006).Cutting et al (2005) and Edmonds (2005) apply a wearlist for iden tifying infection in diabetic foot ulcers. In the checklist, there are four areas under it are the signs and a box opposite to it to check if certain signs are present.Clinical signs of infectionCellulitis- acute inflammation of tissuelymphangitisphlegmon-purulent exudatepus/abscesscrepitus in the jointerythemaincrease in exudates volumelocalised painmalodourprobes to bone up.Systematic signs of infection sicknessFatigueVomitingFeverchillsProbe to bone testbone palpatedno bone involvementWound culturewound dab requiredwound biopsy required.Furthermore, the wound infection continuum of Gary, White, Cooper and Kingsleys (2005 2010) is also use to measure the extinct of infection. It is also a useful adjunct in identification of treatment objectives. The scoring is from 3-0 score of 3 means spreasing infection and 0 is colonised. Moreover, Gray et al (2010) also have the wound exudates continuum it identifies presence of infection since excessive exudates suggest infections.Section 4 . Wound ManagementUsing the same wound feature that you identified in Section 3 critically discuss the different ways there are of managing this problem.Your discussion must includeThe different types of wound care medical medical dressings, products and treatments that could be used to manage this problemOther appropriate/related aspects of patient care such as nutrition and positioningHow the patient experience can be betterNow select one of your identified dressings and answer the questions in the product info table belowProduct information tableName of dressing (the company name)AQUACEL Ag Hydrofiber (Convatec, Hull, UK)Category of dressing (the generic name)Hydrofiber Wound Dressing with Ionic SilverIndications for use subroutine on acute and chorin wounds, including burns, surgical wounds, diabetic foot ulcers, pressure ulcers, and leg ulcersContra-indicationsAquacel Ag Hydrofiber should not be used on individuals who are clarified to or who have had an allergic reaction t o the dressing and its components such as Na Carboymethylcellolose and cash. Its not compatible with oil-based products, such as petrolatum jelly.Sizes useable2x 2, 4x4.7, 6x6, 8x12, 75x18, 39x18Adhesive or non-adhesive?Non-adhesive dressingSecondary dressing needed requiredMoisture retentive dressing such as DuoDERM Extra shrink or VersivaApproximate word count 1000Your answer hereManagement of diabetic foot ulcers are removal of callus, eradication of infection, and drop-off of weight bearing forces, often requiring bed rest with the foot brocaded (Alexander, Fawcett Runciman, 2000).A large proportion of patients with diabetic foot ulceration will develop infection, including osteomyelitis or bone infection and gangrene (OMeara et al, 2006 McIntosh, 2007). An give diabetic ulcer needs immediate medical attention. Jude (2007) stated that infection is a major factor that delays wound healing of a diabetic ulcer. It may be necessary to undertake surgical debridement and drainage of pus. Then a wound swab will be taken from the floor of the ulcer after the callus has been removed. A culture of the excised tissue may result more accurate information (Watkins, 2003). This will help identify the morbific microorganisms and the appropriate antibiotic therapy to be given (Alexander et al., 2000).According to Watkins (2003) patients with superficial ulcer infection can be treated with oral antibiotics such as amoxicillin, flucloxacillin and metronidazole. Since the most likely organisms to infect superficial ulcer are staphylococci, streptococci, and sometimes anaerobes. For patients with deep infections should be hospitalised and started on broad-sprectrum antibiotics. Surgical debridement should then be carried out, which should include all the devitalised tissues, sloughed tendons, and give bones.Jude (2007) said that diabetic foot ulcers generally have multiple organisms isolated from within the wound and methicillin-resistant Staphylococcus aureus (MRSA) is an important vancomycin and teicoplanin can be given to patients infected with MRSA. Anyhow, linezolid can be an secondary which can be administered orally.Furthermore, various local germicides, antiseptics, and antibiotics have been used also in treating infected diabetic foot ulcer. Topical antibiotics like neomycin, bacitracin, neomycin, gentamycin, polymyxin B, mupiricin, fusidic acid, and topical antiseptics are also used in infected foot ulcers. Although antiseptics and antibiotics are widely used, there is insufficient evidence for their use in diabetic foot ulcers (Jude, 2007). Lipsky, Holroyd Zasloff (2008) studies showed that pexiganan cream can be used as an effective alternative to oral antibiotic therapy in treating mildly infected diabetic foot ulcer and might decrease the risk of selecting antimicrobial-resistant bacteria.Dressings also toy an important role in managing infected diabetic foot ulcers. at that place are various dressings available in the market nowadays. The selection of a dressing will depend on the condition of the ulcer. Most infected diabetic foot ulcers produce copious amount of exudates and pus. Dressings are used to control exudates, maintain a moist wound healing environment and eradicated the microorganisms that cause infection. The appropriate dressings for infected wounds with exudates are foam, alginates, hydrofiber, and hydrocolloids that are combined with specie ion. Silver has been shown to have bactericidal gracefulties and has been used in wounds as an antimicrobial for more than century. It acts by impairing the bacterial electron transport system and some of its DNA function. It kills the microbes on contact through multiple mechanism of action, such as inhibiting cellular respiration, denaturing nucleic acids, and altering cellular membrane permeability. Nowadays, Silver ions have been incorporated in hydrofiber, foam, hydrocolloid, and alginate dressings (Bergin Wraight, 2006).Concreet Foam dressin gs with silver can be used during inflammatory phase following debridement and desloughing. It also kills microorganisms on the wounds. It is very sorbefacient that can be left undisturbed for 3-4 days. However, it can cause a drying effect on the wound if there are too minute exudates.Hydrofiber dressings such as Aquacel Ag absorb the exudates, protecting the edges of the wounds from maceration at the same time kills the bacteria in the wound. Alginate dressings is use as a primary dressing and for packing wound, it is good for deeply ulcerated wound with high exudates. Another dressing is hydrocolloid it is best use on wounds with granulating and epithelialising wounds that with low to moderate amounts of exudates.The primary dressings therefore should be either foam-based such as Contreet foam (ColoplastHumlebaek, Denmark) or hydrofiber AQAg (Aquacel Ag Convatec, Chester,UK), both of which will absorb the exudates. A moisture retentive dressing can be used as a secondary dressi ng such as DuoDERM Extra trim down or Versiva. One advantage with the hydrofiber dressing is its capacity to hold wound exudates and microorganisms within its fibres where the bacteria are then eradicated by the ionic silver (Jude, 2007). In addition, the used of hydrofiber dressing in exudating wounds has been proven with research. Jude also implied to purifyd outcomes in infected diabetic foot ulcers and ulcers that are colonised, one should weigh silver dressings as an essential adjunct to wound care to improve its wound bed and to facilitate healing. Studies also revealed that patients treated with AQAg primary dressing showed improved healing and more overall ulcer improvement with less deterioration in the ulcer.Once the acute situation has resolved it will be necessary to ensure redistribution of the weight-bearing forces on the vulnerable foot by the use of specially constructed shoes or moulded insoles. Application of a thoroughgoing contact plaster cast, lightweight sc otch cast boot, or air cast boots may help healing. These conform to the contours of the foot, thereby reducing prune forces on the plantar surface. Great care must be taken, especially with the fitting of plasters, to prevent chafing and subsequent ulcer formation elsewhere on the foot or ankle (Watkins, 2003). If recurrence of neurophatic ulceration is to be avoided, secureness follow-up by a chiropodist will be required. An on-going podiatry to remove excess callus and provide nail care regular assessment, look for active lesions and treat immediately, detect and manage deformities, callus, skin cracks, and discoloration, simple sensory test, examine pulses such as dorsalis pedis and posterior tibial, assess ankle reflex and assess other sensory modalities (Alexander, Fawcett Runciman, 2000).The patients experience will be improved by having a highly structured care. The patients infected ulcer shows healing improvement and prevent from amputation. Understanding the diabetic foot, the proper examination of the patients feet, investigations to classify the foot ulcers, and proper management techniques using a team approach, along with preventive steps, will go a long way in limb salvage and prevention of foot amputation (Pendsey, 2010).Section 5. Evidence based guidanceIdentify a contemporary source of evidence based guidance (i.e. a clinical guideline) which could be used as a basis for providing a high standard of care to patients with this type of wound. Critically discuss how the guidance given in this document might influence your nursing practice including whether you believe there are any omissions or recommendations made that would be difficult to manage in your own clinical setting (Phillipines). You must clearly state the full reference of your chosen guideline document and link your work to other healthcare literature where appropriate.Approximate word count 500Your answer here